FLUORIDES AND KIDNEY DAMAGE – Australia, USA, and Sri Lanka

Indigenous Australians have smaller kidneys
than non-indigenous Australians. Death rates from
chronic kidney disease are 7  to 11 times higher for
non-indigenous males and females respectively.

Fluoride Is A Developmental Nephrotoxin –
Coming To A Kidney Near You

F. & Kidney Dam. s

Indigenous Aus. s

image-of-kidney

People With Kidney Impairment Are At Greater Risk From Fluorides.

Indigenous Australians have smaller kidneys than non-indigenous Australians…
 – A reduced number of nephrons and decreased renal reserve –
(Singh G, White A, Spencer J, Wang Z, Hoy W [1999]).

Indigenous Australians are 6 times more likely than other Australians to be receiving dialysis or to have had a kidney transplant. Death rates from chronic kidney disease are 7 and 11 times as high for non-indigenous males and females respectively.

Australia’s peak medical advisory body, the Australian Government’s National Health & Medical Research Council (NHMRC) advised in their Australian Drinking Water Guidelines 2004 that:

“People with kidney impairment have a lower margin of safety for fluoride intake. Limited data indicates that their fluoride retention may be up to three times normal”

Indigenous-A-ss

Indigenous Australians have high rates of kidney disease and also diabetes, (6 times higher than for non-Aboriginal Australians) which can lead to kidney disease, they are less able to cope with fluoridation, they will have higher plasma fluoride levels, and circulating for longer will accumulate more fluoride in their bodies (particularly bones) because of their decreased ability to excrete fluoride.

BAD YINGAY

The Queensland Government’s proposal to fluoridate indigenous communities, some of which are currently using underground water of untested levels of existing natural fluoride, can be described as reckless stupidity – genocidal, and needs to be reversed immediately. [ Less polite language may be valid. ]

Inorganic fluoride. Divergent effects on human proximal tubular cell viability. R. A. Zager and M. Iwata. Fred Hutchinson Cancer Research Center, Seattle, WA…

Inorganic fluoride. Divergent effects on human proximal tubular cell …

Fluoridation and Renal Disease in Aboriginal and Torres Strait Islanders

 

Fluorides are filtered from the blood by the kidneys and excreted in the
urine. Renal function is quite important and may be closely related to the
differences of susceptibilities to F toxicity in the general population.
F may accumulate more readily in individuals who have impaired 
renal function and, therefore, affects them more seriously. 

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March 1969

U.S.A. SURGEON GENERAL WARNS
AGAINST FLUORIDATED WATER IN KIDNEY MACHINES 

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THE NATIONAL KIDNEY FOUNDATION (USA)
NO LONGER ENDORSES FLUORIDATION:

“The 1981 NKF position paper on fluoridation is outdated.
The paper is withdrawn and will no longer be circulated,
effective from the 10/06/07″ – NKF Board of Director’s meeting.

http://ndt.oxfordjournals.org/cgi/content/full/gfm663v1

❝ The safety margin for exposure to fluoride by renal patients is unknown, measurements of fluoride levels are not routine, the onset of skeletal fluorosis is slow and insidious, clinical symptoms of this skeletal disorder are vague, progression of renal functional decline is multifactorial and physicians are unaware of side effects of fluoride on kidneys or bone. ❞

The Nephrology Dialysis Transplantation is a monthly peer-reviewed medical journal. It is published by Oxford on behalf of the European Renal Association (ERA) and European Dialysis and Transplantation Association (EDTA). It is abstracted and indexed in PubMed/MEDLINE/Index Medicus. According to the Journal Citation Reports, the journal has a 2011 impact factor of 3.564.[1] The journal’s current editor-in-chief is Carmine Zoccali from Italy.

Haemodialysis-Risks Associated With Water Contamination.

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Afican Americans f

Water Fluoridation Targets Black Americans
Wednesday, 12 May 2010

WATER FLUORIDATION
TARGETS BLACK AMERICANS

Silicofluorides and Higher Blood Lead

At present, U.S. public water systems serving over 140 million people are fluoridated with 200,000 tons of commercial grade hydrofluosilicic acid (H2SiF6) and sodium silicofluoride (Na2SiF6), together called “silicofluorides” (or “SiFs”).

Data from numerous studies show that, taking economic, social and racial factors into account, where silicofluorides are used, children absorb more lead from the environment, and there are higher rates of diseases and behavioral problems associated with lead poisoning (including hyperactivity, substance abuse, and violent crime).

Although some early studies showed differences between sodium fluoride and sodium silicofluoride, to this day the substitution of silicofluorides in public water treatment facilities HAS NEVER BEEN SUBJECTED TO APPROPRIATE ANIMAL OR HUMAN TESTING. Recently, the Assistant Administrator of the EPA admitted to Congress that his agency had no data on SiF toxicity and the Chief of the Treatment Technology Evaluation Branch at the National Risk Management Research Laboratory confirmed that the EPA has “no” data on the “health and behavioral effects of fluosilicic acid.”

Despite claims of safety by oral health officials, laboratory research in Germany revealed that silicofluorides do not dissociate completely and have important biological effects. To follow up on this issue, we have compared children’s blood lead levels in communities using SiF treated water with communities using sodium fluoride or with non-fluoridated water. In three separate samples, totaling over 400,000 children, SiF treated municipal water is ALWAYS significantly associated with increased blood lead levels in children.

This effect was evident in a Massachusetts survey of lead levels in 280,000 children (see graph for children exposed to SiF from the Greater Boston water system, from towns that add SiF locally, or from communities using sodium fluoride, and towns without fluoridation). For the state of New York, data was available on venous blood lead levels for 151,225 children in communities of 15,000 to 75,000. Controlling for other factors associated with higher blood lead, silicofluorides were again significantly associated with higher uptake of lead from the environment. For black children, who are especially at risk for high blood lead, those in towns using SiF were less likely to have low blood lead and more likely to have lead over 10µg/dL. To confirm that these results are not due to other socio-economic or demographic factors, additional statistical tests were run.

The third study concerned children’s blood lead levels in the National Health and Nutrition Evaluation Survey (NHANES III), which had reports for 7224 children from 80 counties with populations over 500,000. Since only 4 of these counties had any communities that used sodium fluoride, analysis of the NHANES III data focused on the percentage of the entire county population exposed to silicofluoride treated water.

Among the 1543 children of all ages from large urban counties with over 80% of the population exposed to fluoridation (almost all of whom receive water treated with SiF), average blood lead was 5.12 µg/dL whereas the average for 1139 children in low fluoride exposure counties was 3.64 µg/dL Blood lead in the 473 children sampled from the medium fluoridation counties was 3.23 µg/dL, which was significantly different from the high fluoridation counties but not from either low fluoridation counties or those with unknown fluoridation status, where average blood lead levels were 3.16 µg/dL (S.D. 2.83). Controlling for the Poverty, the effect of SiF use was highly significant (p < .0001). When the sample is divided by age and race, these findings provide six separate samples in which SiF is associated with high blood lead (see Graphs).

In all three populations studied, those children in each racial category and each age group who were highly likely to be exposed to silicofluorides differ strongly in levels of blood lead from those not exposed.

This conclusion was further checked by analyzing available data for health and behavioral traits that have been associated with high blood lead (such as violent crimes, cocaine use and asthma). In each case, those exposed to silicofluoride treated water were more likely to have behavioral or health problems that are more likely among those with high lead in their bodies.

The injection of silicofluorides in public water supplies is a practice whose elimination could possibly contribute to reduced rates of learning disabilities, substance abuse, violent crime, and asthma (all connected with lead poisoning and other toxins). Whatever the benefits to teeth (and this is highly controversial), our research shows that the issues facing the public concern silicofluoride chemistry, toxicology, and the linkage of neurotoxins with behavior or health. Before SiF chemicals are used, citizens must know that they are safe for all.

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For more information, see:

Dartmouth Researcher Warns of Chemicals Added to Drinking Water

(Note: from this [ABOVE] site, that one can download an English translation of Westendorf’s studies of silicofluorides, which have not hitherto been available in the U.S.)

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Research From Sri Lanka

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Fluoride In Drinking Water & Diet: The Causative Factor Of
Chronic Kidney Diseases In The North Central Province of Sri Lanka

  HERE  

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 FLUOROSIS – AND WAYS TO CONTROL IT – India

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Fluoridation & Renal Disease in Aboriginal & Torres Strait Islanders

 

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